Paige Chavers DNP, ACNP-BC, MSPH: a disease state characterized. COPD (CHRONIC PULMONARY OBSTRUCTIVE DISEASE) by SUKHERA. 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Citation: Gundry S (2019) COPD 1: pathophysiology, diagnosis and prognosis. Awareness and compliance with Guidelines for the management of Stable COPD in Current Outpatient practices and patients' knowledge of COPD - Active Detection of Chronic Obstructive Pulmonary Disease and Asthma in the General Population Vol 158. pp 1730 1738, 1998. Remember to update it at least every six months. COPD - Physical Findings. We've encountered a problem, please try again. You will also learn useful health and safety tips for managing your COPD. RN, BSN, PHN. Ventolin) MDI or nebulizer preferred Anticholinergics (e.g. The COPD Pocket Consultant Guide (PCG) app is built to support patients with COPD and their family members in self-management and to assist health care professionals in providing optimal care. Unlock this template and gain unlimited access, Are you already Premium? B - Background. 10. COPD is the progressive and partially reversible Barnett, M. (2008). diagnosis initial investigation, COPD - . Now customize the name of a clipboard to store your clips. PowerPlugs is a trademark of CrystalGraphics, Inc. COPD COMPLETE POWER POINT AS PER GOLD. chronic obstructive pulmonary disease in equine, COPD (Chronic obstructive Pulmonary Disease) PowerPoint Presentation -aslam, Chronic obstructive pulmonary disease (copd) power point, Nursing care plans, concept map bronhial asthma, Introduction & investigations to respiratory diseases, L'Docile - Respiratory diseases & nebulization Report, J. Parker Emphysema Presentation Powerpoint, COPD - Chronic Obstructive Pulmonary Disease |medico X| Pathology. PowerPoint Presentation Last modified by: This booklet will help you plan for emergencies and stay safe during a disaster event. A widowed, 60-year-old, retired post office clerk, her main complaint is breathlessness after moderate exertion. Do not sell or share my personal information, 1. 28-7, Emphysema Pathophysiology Hyperinflation of alveoli Destruction of alveolar walls Destruction of alveolar capillary walls Narrowed airways Loss of lung elasticity, Emphysema Pathophysiology Two types: Centrilobular (central part of lobule) Most common Panlobular (destruction of whole lobule) Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are: Hyperinflation of alveoli Destruction of alveolar capillary walls Narrowed, tortuous small airways Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of Mucus Smooth muscle spasm Inflammatory process Collapse of bronchiolar walls Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed Air goes into the lungs but is unable to come out on its own and remains in the lung Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention As more alveoli coalesce, blebs and bullae may develop Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion Compensation is done by increasing respiratory rate to increase alveolar ventilation Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations Dyspnea Progresses in severity Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations Patient becomes chest breather, relying on accessory muscles Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are: Hyperplasia of mucus-secreting glands in trachea and bronchi Increase in goblet cells Disappearance of cilia Chronic inflammatory changes and narrrowing of small airways Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation Primary pathologic mechanism causing changes Narrow airway lumen and reduced airflow d/t hyperplasia of mucus glands Inflammatory swelling Excess, thick mucus, Chronic Bronchitis Pathophysiology Greater resistance to airflow increases work of breathing Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion Tendency to hypoventilate and retain CO2 Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions Bronchospasm frequently develops More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms: Frequent, productive cough during winter Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing Cough Dyspnea on exertion History of smoking Normal weight or heavyset Ruddy (bluish-red) appearance d/t polycythemia (increased Hgb d/t chronic hypoxemia)) cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis) Cor pulmonale (Rt heart hypertrophy + RV failure) Pneumonia Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities History and physical exam Pulmonary function studies reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs PaO2 PaCO2 (especially in chronic bronchitis) pH (especially in chronic bronchitis) Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy Bronchodilators as maintenance therapy -adrenergic agonists (e.g. The SlideShare family just got bigger. Download Presentation. We've updated our privacy policy. 3. possible areas to cover. COPD - . Sadness, tiredness, hopelessness and helplessness, social withdrawal and interpersonal problems, sleep and eating problems. This flyer will give you helpful tips and tools for staying healthy, avoiding illness, and recognizing early warning signs of an infection or flare-up. Chronic Bronchitis Tachypnea Accessory respiratory muscle use. review. 527 Views Download Presentation. chronic bronchitis emphysema. definition epidemiology risk, COPD - . Updated November 2021. Chronic obstructive pulmonary disorders COPD. This article has been double-blind peer reviewed Scroll down to read the article or download a print-friendly PDF here (if the PDF fails to fully . Course Hero is not sponsored or endorsed by any college or university. michele ritter, m.d. He states he has been out of his medications for about 3, This week's content addresses common techniques and testing that can be prescribed by the Nurse Practitioner. within 30 Days Required ; chronic obstructive pulmonary disease by: jesse and courtney. Energy-conserving tips are presented. chronic obstructive pulmonary disease. 21 slides. Contact Hours: 42.5 This nursing continuing professional development activity was approved by the American Association of Critical-Care Nurses, an accredited approver by the American Nurses Credentialing Center's Commission on Accreditation. possible areas to cover. It has a simple style, with a white background and light blue waves and . You can read the details below. Tachypnea Accessory respiratory muscle use, COPD Hallmark symptom - Dyspnea Chronic productive cough Minor hemoptysis pink puffer blue bloater. COPD Description Characterized by presence of airflow obstruction Caused by emphysema or chronic bronchitis Generally progressive May be accompanied by airway hyperreactivity May be partially reversible. Chronic Obstructive Pulmonary Disease (COPD) Chronic obstructive pulmonary disease (COPD), which includes chronic bronchitis and emphysema, is a long-term lung disease that makes it hard to breathe. This booklet covers many important topics, such as how bronchiectasis is diagnosed, treatment options, tips for living well and reducing exacerbations, and how to find support for living with bronchiectasis. , Laboratory Tests Elevated hematocrit suggests chronic hypoxemia. . Seventh National Doctors of Nursing Practice Conference Poster Presentations Click the title of the presentation to view the poster in PDF. 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